Payment & Shipping Terms:
|State:||White Powder||Product Name:||Nicotinamide Mononucleotide|
|Advantages:||Reship Policy||Suitable For:||Adult|
|Ship Way:||EMS,HKEMS,Fedex,DHL, HKPOST||CAS:||1094-61-7|
|Other Name:||NMN||Other Name1:||BETA-NICOTINAMIDE MONONUCLEOTIDE|
1. Basic Info
β-NMN Beta-Nicotinamide Mononucleotide
β-NMN CAS: 1094-61-7
β-NMN MF: C11H15N2O8P
β-NMN MW: 334.22
β-NMN EINECS: 214-136-5
β-NMN storage temp. 2-8°C
β-NMN Chemical Properties: White powder.
β-NMN Payment:T/T, Western Union and Money Gram
β-NMN Delivery:Within 24hours After Your Payment
β-NMN Express: EMS, DHL, FedEx, etc(door-to-door)
|Synonyms||Nicotinamide mononucleotide; Nicotinamide ribonucleoside 5'-phosphate; Nicotinamide ribonucleotide; Nicotinamide ribotide; beta-D-NMN; beta-NMN|
|CAS Registry Number||1094-61-7|
Beta-Nicotinamide mononucleotide (NMN) is an intermediate of NAD+ biosynthesis produced from nicotinamide through nicotinamide phosphoribosyltransferase (NAMPT) and also referred to as NMN or nicotinamide ribotide. In recent studies of NMN applications to prevention and diagnosis of age-dependent diseases such as diabetes, neurodegenerative disorder, cardiac disease etc., it has become one of hottest biomarkers on discussion for anti-aging, lately. As one of worldwide leading manufacturers is now providing highly qualified fully physiologically active, beta-Nicotinamide mononucleotide (NMN) as to support basic research in not only Academia but also Pharmaceuticals.
The coenzyme NAD and its derivatives are involved in hundreds of metabolic redox reactions and are utilized in protein ADP-ribosylation, histone deacetylation, and in some Ca2+ signaling pathways. NMNAT (EC 184.108.40.206) is a central enzyme in NAD biosynthesis, catalyzing the condensation of nicotinamide mononucleotide (NMN) or nicotinic acid mononucleotide (NaMN) with the AMP moiety of ATP to form NAD or NaAD.
NMNAT1 is the most widely expressed of three orthologous genes with nicotinamide-nucleotide adenylyltransferase (NMNAT) activity. Genetically engineered mice lacking NMNAT1 die during early embryogenesis, indicating a critical role of this gene in organismal viability. In contrast, mice lacking NMNAT2, which is expressed predominantly in neural tissues, complete development but die shortly after birth. However, NMNAT1 is dispensable for cell viability, as homozygous deletion of this gene occurs in glioblastoma tumors and cell lines. NMNAT enzymatic activity is probably essential at the cellular level, as complete ablation of NMNAT activity in model organisms leads to cellular inviability.
|Pain Killer Product||Cas No|
|Procaine HCL(10-40 mesh)||51-05-8|
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